![]() ![]() This idea implies that these two phenomena – insulin resistance and beta cells dysfunction – occur for entirely separate reasons. Conventional medical wisdom holds that this dysfunction occurs because of exhaustion and eventual scarring of the insulin-producing cells. Furthermore, only insulin resistance and virtually nothing else causes beta cells dysfunction. Without dietary intervention, this insulin resistance almost always leads to the second problem, beta cell dysfunction. This compensatory hyperinsulinemia forces the glucose into the cells, keeping blood glucose levels normal. Insulin resistance develops early in the disease process, typically preceding the diagnosis of type 2 diabetes by a decade or more, but blood glucose remains relatively normal because the pancreatic beta cells increase insulin production to balance. ![]() First, insulin resistance, an overflow phenomenon, is caused by fatty infiltration of the liver and muscle. While type 2 diabetes is considered primarily a disease of excessive insulin resistance, it actually represents two separate physiological defects. ![]()
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